{"id":178,"date":"2024-09-30T00:28:00","date_gmt":"2024-09-30T00:28:00","guid":{"rendered":"https:\/\/word.steevenson.fr\/?p=178"},"modified":"2024-09-30T00:33:42","modified_gmt":"2024-09-30T00:33:42","slug":"twop-sik-nan-san-hyperglycemie","status":"publish","type":"post","link":"https:\/\/word.steevenson.fr\/index.php\/2024\/09\/30\/twop-sik-nan-san-hyperglycemie\/","title":{"rendered":"Tw\u00f2p sik nan san( hyperglyc\u00e9mie)"},"content":{"rendered":"\n

Sik<\/strong>, oubyen “glucose<\/strong>“( glikoz<\/strong>), Mezanmi., eskenn konnen nou manje sik <\/strong>chak jou sou tout f\u00f2m<\/strong>? Si yo mande nou ki val\u00e8 sik <\/strong>nou konsomen<\/strong> pa jou \u00e8ske nap konnen? Gen moun<\/strong> ki panse ke yo pa konsomen<\/strong> anpil sik(glucose<\/strong>) paske yo pa manje bagay ki dous<\/strong>, Eske se vre? <\/p>\n\n\n\n

Younn nan rezon <\/strong>nou souvan pa konnen kantite <\/strong>sik nou konsome<\/strong> pa jou, se paske Anpil fwa l\u00e8 nou tande sik(glikoz) nou panse souvan av\u00e8k sik <\/strong>de tab lan (sik rafine) oubyen nou panse ak sik ki nan manje ki dous<\/strong> tankou kann ak mango <\/strong>ak sir\u00e8t. Men an reyalite sik(glikoz<\/strong>) kache preske nan tout manje<\/strong>, espesyalman nan aliman ke yo rele “hydrate de carbone<\/strong>“( kaboyidrat<\/strong>), yon sous manje ki gen anpil sik ki gen lamidon<\/strong>( Kaboyidrat<\/strong>) ki se yon fom sik pw\u00e8s<\/strong> ki nan yanm, p\u00f2mdete<\/strong> ak bannann<\/strong>, labapen<\/strong>, diri,<\/strong> mayi<\/strong>, ble,<\/strong> farin, bonbon<\/strong> e latriye<\/p>\n\n\n\n

Nan sans sa, nou klere<\/strong> je nou pou konprann<\/strong> ke on manje ki pa dous <\/strong>pa vle di kel pa gen sik(glikoz)<\/strong>. Alekile, Le f\u00e8t<\/strong> ke anpil moun pa enfome<\/strong> sou val\u00e8 fason sik(glikoz) ka egziste nan manje<\/strong>, li vin f\u00e8 ke anpil moun ap konsomen on val\u00e8 sik ki depase kantite sist\u00e8m <\/strong>yo bezwen chak jou. Piske nap viv nan yon tan<\/strong> ki fokis sou f\u00e8 <\/strong>manje fasil a baz de sik( glikoz)<\/strong> pou elimine grangou <\/strong>rapid. Af\u00e8 sik lan vin tounnen yon gwo dange sosyal<\/strong> pou kantite dega “twop sik nan san”(hyperglyc\u00e9mie<\/strong>) ap f\u00e8 nan la sante piblik<\/strong>.<\/p>\n\n\n\n

<\/p>\n\n\n\n

Pou t\u00e8t sa<\/strong>, li enpotan pou nou konnen Kisa twop sik nan san<\/strong> vle di? Eske twop sik nan san<\/strong> se yon maladi<\/strong>? E Ki fason on moun vin <\/strong>gen twop sik nan san<\/strong>? ki kalite moun ki afekte <\/strong>plis? kijan nou ka evite<\/strong> gen twop sik nan san<\/strong>? Ki konsekans twop sik nan san <\/strong>genyen nan sitem nou<\/strong>? Ki sentom<\/strong> moun ki gen anpil sik nan san genyen<\/strong>? eske twop sik nan san ka trete ? <\/p>\n\n\n\n

KISA TWOP SIK NAN SAN VLE DI?<\/strong><\/p>\n\n\n\n

N\u00f2malman, tout selil(cellules<\/strong>) nan ko nou bezwen sik(glikoz<\/strong>) kom en\u00e8ji poul fonksyone sitou selil ki nan s\u00e8vo<\/strong> nou ak selil nan k\u00e8<\/strong> nou. Pou tet sa s\u00e8vo nou ak k\u00e8 nou itilize sik 24 sou 24<\/strong>,  konsa nou toujou gen yon kantite sik nomal kap sikile nan san nou ant 77 a 99 miligram pa desilit.<\/strong> Chak le nou manje, lestomak<\/strong> nou etilize asid<\/strong> pou dekonpoze manje an poul jwenn eneji <\/strong>sou fom sik(glucose<\/strong>). Le sik sa rantre nan san nou li monte kantite sik lan pi wo ke 99<\/strong>, tankou 180<\/strong> oubyen 300,<\/strong> depandan de kantite sik<\/strong> manje an genyen. Rapidman gen yon \u00f2gann<\/strong> ki rele “pancreas<\/strong>” ( pankrea<\/strong>), depil we sik lan monte <\/strong>nan san nou li pwodwi yon \u00f2m\u00f2nn<\/strong>(sibstans) ki rele insuline<\/strong> (Ensilin<\/strong>). Ensilin<\/strong> lan sevi k\u00f2m kle<\/strong> pou ouv\u00e8 p\u00f2t selil yo pou nou ka itilize sik lan kom en\u00e8ji<\/strong>. Espesyalam nan s\u00e8vo<\/strong> ak an k\u00e8<\/strong> nou, epi tout javr\u00e8t<\/strong>( muscles) k\u00f2 nou.<\/p>\n\n\n\n

Travay pankrea <\/strong>se pwodwi ensilin<\/strong> a trav\u00e8 “selil Beta<\/strong>” ( cellules Beta) chak le nap manje<\/strong> pou li ka desann <\/strong>e kenbe sik nan<\/strong> nivo ki pa danje pou ko nou. Gen sitiyasyon kote moun manje tw\u00f2p kabohidrat<\/strong>( lamidon) ki fe sik la toujou wo nan san<\/strong> chak jou jiskaske selil <\/strong>nan ko nou vin<\/strong> fe rezistans ak Ensilin<\/strong>, le konsa ensilin paka ede sik<\/strong> lan antre nan selil s\u00e8vo <\/strong>ak k\u00e8<\/strong> ak jevr\u00e8t<\/strong>. sik la kounya ki pa jwenn<\/strong> kote poul ale li rete ap sikile<\/strong> nan sa ak yon nivo<\/strong> ki byen wo <\/strong>e ki danjere<\/strong>, le konsa nou etabli on kondisyon <\/strong>yo rele sik wo<\/strong> oubyen tw\u00f2p sik nan san (hyperglyc\u00e9mie<\/strong>) ki ka abouti a “diabetes<\/strong>“( diab\u00e8t<\/strong>), maladi sik<\/strong>. <\/p>\n\n\n\n

KI FASON ON MOUN VIN GEN TWOP SIK NAN SAN?<\/strong><\/p>\n\n\n\n

K\u00f2m<\/strong> nou konnen Pankrea<\/strong> jwe on r\u00f2l enpotan nan kontrole<\/strong> e kenbe kantite <\/strong>sik kap sikile<\/strong> nan san nan val\u00e8<\/strong> n\u00f2ma<\/strong>l. Pout\u00e8t sa pif\u00f2<\/strong> pwobl\u00e8m<\/strong> tw\u00f2p<\/strong> sik nan san gen pou w\u00e8 av\u00e8k yon pwobl\u00e8m<\/strong> ki egzizte nan pankrea<\/strong> . Konsa gen 5<\/strong> fason nou ka fe sik tw\u00f2 wo(diab\u00e8tes<\/strong>).<\/p>\n\n\n\n

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  1. Premye fason<\/strong> malerezman se moun ki fet ak yon pankrea<\/strong> ki pa ka pwodwi ensulin<\/strong>, oubyen on pankrea ki pa pwodwi ase insulin<\/strong>. Yo rele sa dyab\u00e8t tip 1<\/strong>. Kisa yon tip de tw\u00f2p sik nan san<\/strong> ki egziste depi l\u00e8 timoun <\/strong>nan f\u00e8t. Kote ke selil sist\u00e8m<\/strong> defans( sist\u00e8m iminit\u00e8<\/strong>) ko nou atake “silil Beta” <\/strong>nan pankrea<\/strong>. ki fe ke “selil Beta<\/strong>” yo mouri <\/strong>nan pankrea<\/strong> epi yo poko pwodwi ensilin<\/strong>. Tip diabet sa<\/strong> rele insilin depandan<\/strong>, sa vle di Moun <\/strong>sa bezwen pran piki ensilin <\/strong>pou tout lavi<\/strong> li. Moun sa konn<\/strong> devlope “pied de charcot<\/strong>” kote afketasyon n\u00e8<\/strong> yo ak zo <\/strong>atikilasyon pye<\/strong> moun nan. <\/li>\n\n\n\n
  2. Dezy\u00e8m<\/strong> fason moun ka f\u00e8 tw\u00f2p sik nan san<\/strong>, se l\u00e8 ke pankrea<\/strong> travay tw\u00f2p le yon moun gen yon rejim alimante<\/strong> ki rich ak kaboyidrat, <\/strong>nou telman pwodwi anpil ensilin pou ranje sik nan san jiskaske<\/strong> sist\u00e8m nou devlope rezitans ak ef\u00e8 ensilin<\/strong> nan selil yo<\/strong>. Yo rele sa dyab\u00e8t tip 2<\/strong>, ki se yon dyab\u00e8t kote ke ensilin lan paka ouv\u00e8 pot selil yo <\/strong>pou sik ka antre<\/strong>, konsa sik<\/strong> lan akimile nan san <\/strong> rive jiska<\/strong> 800 d\u00e8fwa. Dyab\u00e8t Tip 2 an pi komen kay granmoun, e nan ka sa, pankrea pwidwi ensilin<\/strong> men selman nou pa ka itilizel<\/strong> pou konsome <\/strong>sik kom eneji.<\/li>\n\n\n\n
  3. Twazy\u00e8m<\/strong> fason, se kay fi ki ansent, ki gen sik wo <\/strong>pou la premye fwa pandan yo ansent<\/strong> jiskaske yo akouche. Epi malerezman apre yo akouche<\/strong> sik lan toujou rete wo <\/strong>li pa janm desann anko<\/strong>. yo rele sa diab\u00e8t jestasyonal<\/strong> ( diab\u00e8tes gestasionelle).<\/li>\n\n\n\n
  4. katriy\u00e8m<\/strong> tip lan rele “MODY<\/strong>” ( maturity onset diabetes of the young<\/strong>) Piske tip 2 an <\/strong>pi koman kay granmoun pou tet fisiopatoloji an<\/strong>, le yo jwenn li tip 2 an <\/strong>prezante nan j\u00e8nn moun <\/strong>yo rele li “MODY<\/strong>“. MODY<\/strong> an pa <\/strong>monte sik lan wo<\/strong> nan yon kantite<\/strong> ki danjere<\/strong>, paske le pli souvan jenn moun<\/strong> sa gen yon pwofil fizik<\/strong> n\u00f2mal <\/strong>ak pwa n\u00f2mal <\/strong>epi yo pa manifste sentom tipik sik wo<\/strong> nan san.<\/li>\n\n\n\n
  5. Senky\u00e8m<\/strong> tip lan se “LADA<\/strong>” ( late onset diabetes of the adults<\/strong>) Piske diabet tip 1 an koman kay timoun plis, l\u00e8 li prezante kay granmoun nan yon laj avanse yo rele sa “LADA<\/strong>” ki se yon tip de sik wo ki pataje menm fisiopatoloji diabet tip 1 an<\/strong>. Si nou raple nou dyab\u00e8t tip 1<\/strong> an toujou prezante kay timoun <\/strong>men nan ka sa li manifste kay granmoun<\/strong> ak menm karateristik klinik tip 1<\/strong> an. Tankou on bagay ki barew <\/strong>nan wout a yon laj<\/strong> san atann<\/strong>. Se pou t\u00e8t<\/strong> sa yo relel “LADA<\/strong>“.<\/li>\n<\/ol>\n\n\n\n

    ESKE TWOP SIK NAN SAN SE YON MALADI?<\/strong><\/p>\n\n\n\n

    Tw\u00f2p sik<\/strong> nan san (hyperglyc\u00e9mie<\/strong>) pa<\/strong> souvan yon maladi,<\/strong> men si tw\u00f2p sik nan sa <\/strong>pa korije li ka vin konplike a yon maladi ki rele “dyab\u00e8t<\/strong>“, kise yon kondisyon kote tw\u00f2p sik nan san<\/strong> f\u00e8 selil( cellules) yo vin reb\u00e8l ak ensilin, malgre pankrea pwodwi ensilin l\u00e8 nou fin manje, ensilin lan pa ka ouv\u00e8 pot silil yo(membranes<\/strong> cellulaires<\/strong>) pou sik lan ka antre annda selil lan(cytoplasme)<\/strong> poul itilize sik <\/strong>lan. Le kosan sik lan rete nan san <\/strong>lap monte pil sou pil. Le nou cheke sik nan san nou li ka men wote nan 300 a 500 <\/strong>defwal konn piwo 800<\/strong>. Al\u00e8kile, yo rele diabet ki reziste ak ensilin. K\u00f2m globil rouj nan san atache ak glucose <\/strong>sou f\u00f2m HBA1c<\/strong>( Hemoglobine A1c<\/strong>), yo ititilze pousantaj sik <\/strong>ki atache avek globil rouj <\/strong>pou konnen ki kantite sik ki sikile<\/strong> nan san, nivo nomal<\/strong> lan se mwens<\/strong> ke 5.6%.<\/strong> L\u00e8 nivo A1c ant 5.6% a 6.4% <\/strong>yo rele sa Prediab\u00e8tes<\/strong> oubyen atak dyab\u00e8t<\/strong>. Dyagnostik Dyab\u00e8t etabli <\/strong>le nivo A1c an<\/strong> igal ou plus a 6.5%<\/strong>. <\/p>\n\n\n\n

     KI KALITE MOUN KI AFEKTE AK TWOP SIK NAN SAN?<\/strong><\/p>\n\n\n\n

    Moun ki a risk <\/strong>pou devlop<\/strong>e tw\u00f2p sik nan san<\/strong> se moun kap manje anpil kabohydrat<\/strong>, ak pi ki pa f\u00e8 sp\u00f2<\/strong>, moun ki obez<\/strong>, moun ki ap pran steroid<\/strong>, moun ki ansent<\/strong>, moun gen anpil stress<\/strong>, moun ki gen manje<\/strong> depase le yo strese<\/strong>. Moun ki manje twop bagay dous<\/strong>, moun ki gen pwobl\u00e8m<\/strong> nan pankrea<\/strong> direkteman.<\/p>\n\n\n\n

    KIJAN NOU KA EVITE GEN TWOP SIK NAN SAN?<\/strong><\/p>\n\n\n\n

    Glikoz(sik) <\/strong>se tankou gaz <\/strong>li ye pou ko nou. Sa vle di san Glikoz( sik<\/strong>) nou pap gen eneji <\/strong>ak f\u00f2s ke sist\u00e8m nou bezwen pou fonksyone<\/strong>. Antou, se s\u00e8l nan manje nou ka jwenn sik( glikoz<\/strong>) pou nou fonksyone<\/strong>. Se sak fe nou bezwen manje<\/strong> chak jou pou nou ka bay sistem<\/strong> nan sik pou li ka travay<\/strong>. Le nou manje epi nou pa egzesis<\/strong> ko nou konseve sik lan sou f\u00f2m gr\u00e8ss <\/strong>epi on pati nan sik lan rete akimile <\/strong>nan san. Konsa, meye fason pou evite soufri twop sik <\/strong>nan san se chanje rejim alimant\u00e8 <\/strong>nou, manje mwens kaboyidrat<\/strong> epi sak pi enpotan<\/strong> se fe egz\u00e8sis<\/strong> pou ede selil<\/strong> yo itilize sik kap sikile nan san kom en\u00e8ji<\/strong>.<\/p>\n\n\n\n

    KI KONSEKANS TWOP SIK NAN SAN GENYEN NAN SISTEM NOU ?<\/strong><\/p>\n\n\n\n

    konsekans<\/strong> tw\u00f2p sik <\/strong>nan sa apa de dyab\u00e8t<\/strong>, sik la ka akimile<\/strong> tout l\u00f2t kote nan ko<\/strong> an l\u00e8l tw\u00f2 wo nan san<\/strong>.<\/p>\n\n\n\n

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    1. Tw\u00f2p sik <\/strong>nan san<\/strong> ka vin akimile nan fwa an grand kantite<\/strong>, li vin koz enflamayon<\/strong> nan fwa<\/strong> ki rele “Hepatites<\/strong>” ( fwa anflame)<\/strong>, anplis fwa<\/strong> konveti eks\u00e8 sik lan<\/strong> an gr\u00e8s <\/strong>nan fwa ki vin bay on “foie gra<\/strong>s”( gwo fwa<\/strong>).<\/li>\n\n\n\n
    2. Eks\u00e8<\/strong> sik<\/strong> lan ka akimile nan fib optik zye<\/strong> nou, ki ka koz moun<\/strong> vin av\u00e8g<\/strong>. Moun<\/strong> ka devlope katarak<\/strong>.<\/li>\n\n\n\n
    3. Tw\u00f2p sik <\/strong>nan sa ka akimile <\/strong>nan at\u00e8<\/strong> ki nan k\u00e8<\/strong> nou, li ka devlepe blokaj <\/strong>nan tib kadyak<\/strong>( arteres coronaire<\/strong>) ki vin ogmante risk<\/strong> pou fe atak kadyak <\/strong>ak ensifizans kadyak<\/strong>.<\/li>\n\n\n\n
    4. Tw\u00f2p<\/strong> sik nan san ka akimile <\/strong>nan fib n\u00e8vez<\/strong> yo( neuropathy diab\u00e9tique<\/strong>), moun konn pedi sansayon<\/strong> nan pye<\/strong> yo paske n\u00e8 <\/strong>yo domaje<\/strong> ak sik<\/strong>, le konsa yo pa santi<\/strong> doule byen<\/strong>. Moun<\/strong> ka blese nan pye<\/strong> san yo pa janm santi sa<\/strong>. Blese<\/strong> an ke enfekte ak mikw\u00f2b<\/strong>, yo rele sa “pied diabetique<\/strong>” le konsa si enfeksyon an rive nan zo<\/strong>, yo ka oblije koupe pye<\/strong> an oubyen z\u00f2tey nan pye <\/strong>ki afkete an avan li pwopaje nan san<\/strong>.<\/li>\n\n\n\n
    5. Tw\u00f2p <\/strong>sik nan san ka ekimile<\/strong> nan s\u00e8vo<\/strong>, le konsa li ogmante risk <\/strong>pou soufri alzheimer<\/strong>, ak str\u00f2k<\/strong> ak demence<\/strong>( p\u00e8di t\u00e8t<\/strong>).<\/li>\n\n\n\n
    6. Tw\u00f2p sik nan san<\/strong> ki pa kontwole<\/strong>, pwiske ko nou<\/strong> paka itilize sik<\/strong> kom en\u00e8ji<\/strong>, li komanse etilize gr\u00e8s<\/strong> tankou trigliserid<\/strong>( triglycrides<\/strong>) kom sous en\u00e8ji. Pa konsekan fwa<\/strong> pwodwi yon metabolit<\/strong> ki gen menm strikti <\/strong>ak aset\u00f2nn<\/strong>. Metabolikman, Sistem<\/strong> nou pa zanmi ak aset\u00f2nn<\/strong>. Konsa, Tw\u00f2p<\/strong> aset\u00f2nn nan san fe sist\u00e8m<\/strong> nou vin asid<\/strong> e li mete str\u00e8ss<\/strong> nan sist\u00e8m nan<\/strong>, on kondition metabolik<\/strong> ki rele ketoacidose diab\u00e9tique<\/strong> (k\u00f2 moun nan vin asid ak asetonn<\/strong>) ki ka abouti a koma <\/strong>e lanm\u00f2.<\/strong> Kondisyon <\/strong>sa pi komen nan fom dyabet tip 1<\/strong>. Li difisil<\/strong> pou moun rive<\/strong> nan faz<\/strong> fatal sa ak dyab\u00e8t tip 2.<\/strong><\/li>\n<\/ol>\n\n\n\n

      KI SENTOM MOUN KI GEN ANPIL SIK NAN SAN GENYEN?<\/strong><\/p>\n\n\n\n

      Moun<\/strong> ki gen anpil sik nan san<\/strong> ki pa <\/strong>trete prezante yon kad klinik klasik<\/strong> ki rele “l\u00e8 3 P.”<\/strong><\/p>\n\n\n\n

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      1. Polyurie<\/strong> : sa vle moun nan urine souvan<\/strong>, se yon fason pou ko nou ka elimine sik nan pipi nou<\/strong>. Moun sa ka vin dezidrate, bouch<\/strong> li seche epi po<\/strong> li seche<\/strong>.<\/li>\n\n\n\n
      2. Polydipsie<\/strong>: Pwiske moun sa pipi anpil<\/strong>, li f\u00e8 ke li swaf anpil, konsa li bw\u00e8 dlo souvan p<\/strong>oul ka ranplase dlo li pedi nan plede pipi an<\/strong>.<\/li>\n\n\n\n
      3. Polyphagie<\/strong>: moun sa grangou souvan<\/strong>, malgre li f\u00e8k sot manje<\/strong>, paske ko li paka itilize sik lan k\u00f2m<\/strong> en\u00e8ji<\/strong>, s\u00e8vo <\/strong>an panse se sik ki pa gen nan san<\/strong> ase, epi li estimile<\/strong> on dezi egazajere<\/strong> pou manje pou jwenn <\/strong>sik. Pou t\u00e8t<\/strong> sa moun sa souvan<\/strong> pedi pwa, epi li santi<\/strong> li pa gen en\u00e8ji<\/strong>.<\/li>\n<\/ol>\n\n\n\n

        <\/p>\n\n\n\n

        ESKE TWOP SIK NAN SAN KA TRETE?<\/strong><\/p>\n\n\n\n

        Wi, tw\u00f2p sik nan sa<\/strong>( hyperglyc\u00e9mie<\/strong>) ka trete avan li f\u00e8 dega<\/strong>, oubyen avan li vin tounnen dyab\u00e8t<\/strong>. Pousa li dwe dedekte<\/strong> a tan. Yon tes san<\/strong> glikoz nan san ka p\u00e8m\u00e8t ou konnen siw f\u00e8<\/strong> sik wo<\/strong> ou pa. Av\u00e8k yon glikom\u00e8t<\/strong>( machin<\/strong> pou mezire sik rapid<\/strong>) ou ka detekte<\/strong> ki nouvo sik<\/strong> ou ye l\u00e8w<\/strong> fek leve le maten<\/strong>. Nivo n\u00f2mal <\/strong>sipoze ant 70 a 99 ajen<\/strong>. Sil pi wo ke sa ou vle fe yon t\u00e8s san<\/strong> nan laboratwa<\/strong> pouw konnen nivo A1<\/strong>c ki se idatificat\u00e8 dyab\u00e8t<\/em><\/strong>.<\/p>\n\n\n\n

        An rezime<\/strong>, Se pal s\u00e8l manje dous<\/strong> ki ka monte <\/strong>sik nan san, anpil lot manje tankou kabohydrat <\/strong>se ypon gwo akize <\/strong>nan zaf<\/strong>\u00e8 sik nan san. Tw\u00f2p sik nan san<\/strong> se yon pwobl\u00e8m tr\u00e8 komen<\/strong> ki afekte moun <\/strong>nan tout laj<\/strong>. Defwa se yon kondisyon iminit\u00e8 <\/strong>kay timoun <\/strong>ki koz li, ki se dyab\u00e8t tip 1<\/strong>. D\u00e8fwa se fason nou manje kisa rezon<\/strong> ki pi komen<\/strong> ki fe moun devlope konplikasyon tw\u00f2p sik nan <\/strong>sa ki se dyab\u00e8t tip 2<\/strong>. Egz\u00e8sis<\/strong> ak chanjman rejim alimant\u00e8<\/strong> se mey\u00e8 fason pou evite dyab\u00e8t tip 2<\/strong>. Pan setan, pran piki ensilin <\/strong>pou lavi<\/strong> se mey\u00e8 fason <\/strong>pou kontrole diabet tip 1. Manifestasyon klasik sik wo nan san<\/strong>(hyperglicemie<\/strong>) prezante ak “3 P” <\/strong>yo, polyurie<\/em><\/strong>, poly<\/em>d<\/strong>ipsie,<\/strong> polyphagie<\/strong><\/em>, e pami konplikasyon ki grav gen pye dyabetik<\/strong>, avegleman,<\/strong> strok<\/strong>, atak<\/strong> kadyak<\/strong>, p\u00e8di tit<\/strong>, ak konplikasyon nan ren<\/strong> ak fwa<\/strong> ak acidosis diabetique<\/strong> ki ka debouche sou koma ak lanmo<\/strong> espesylaman nan ka dyabet tip 1<\/strong>. <\/p>\n\n\n\n

        <\/p>\n","protected":false},"excerpt":{"rendered":"

        Sik, oubyen “glucose“( glikoz), Mezanmi., eskenn konnen nou manje sik chak jou sou tout f\u00f2m? Si yo mande nou ki val\u00e8 sik nou konsomen pa jou \u00e8ske nap konnen? Gen moun ki panse ke yo pa konsomen anpil sik(glucose) paske yo pa manje bagay ki dous, Eske se vre?  Younn nan rezon nou souvan pa […]<\/p>\n","protected":false},"author":2,"featured_media":0,"comment_status":"open","ping_status":"open","sticky":false,"template":"","format":"standard","meta":{"footnotes":""},"categories":[1],"tags":[],"class_list":["post-178","post","type-post","status-publish","format-standard","hentry","category-uncategorized"],"_links":{"self":[{"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/posts\/178","targetHints":{"allow":["GET"]}}],"collection":[{"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/posts"}],"about":[{"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/types\/post"}],"author":[{"embeddable":true,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/users\/2"}],"replies":[{"embeddable":true,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/comments?post=178"}],"version-history":[{"count":6,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/posts\/178\/revisions"}],"predecessor-version":[{"id":184,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/posts\/178\/revisions\/184"}],"wp:attachment":[{"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/media?parent=178"}],"wp:term":[{"taxonomy":"category","embeddable":true,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/categories?post=178"},{"taxonomy":"post_tag","embeddable":true,"href":"https:\/\/word.steevenson.fr\/index.php\/wp-json\/wp\/v2\/tags?post=178"}],"curies":[{"name":"wp","href":"https:\/\/api.w.org\/{rel}","templated":true}]}}